Search for publications in the Transfection Database with Polyplus transfection reagents or for transfection conditions.

Over 6000 publications, 1000 cell lines and primary cells available.

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Found 5890 results :
Cell Linein vitro
in vivo
Delivered MoleculeReagentResults & Citations
HEK-293in vitroDNAjetPEI
Orth, J. H. et al. (2005)

J Biol Chem 280, 36701-7
Pasteurella multocida toxin-induced activation of RhoA is mediated via two families of G{alpha} proteins, G{alpha}q and G{alpha}12/13
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B16-F10in vitro / in vivoDNAin vivo-jetPEI, jetPEI
Ortiz, R. et al. (2009)

J Mol Med 87, 899-911
The cytotoxic activity of the phage E protein suppress the growth of murine B16 melanomas in vitro and in vivo
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MG-63, OSRGA, Saos-2in vitrosiRNAINTERFERin
Ory, B. et al. (2008)

J Cell Mol Med 12, 928-41
Farnesyl diphosphate synthase is involved in the resistance to zoledronic acid of osteosarcoma cells
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L6in vitrosiRNAjetPRIME
Osorio-Fuentealba, C. et al. (2013)

Diabetes 62, 1519-26
Electrical stimuli release ATP to increase GLUT4 translocation and glucose uptake via PI3Kgamma-Akt-AS160 in skeletal muscle cells
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THP-1in vitrosiRNAINTERFERin
Osto, E. et al. (2008)

Circulation 118, 2174-82
Inhibition of protein kinase Cbeta prevents foam cell formation by reducing scavenger receptor A expression in human macrophages
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HUVECin vitroDNAjetPEI-HUVEC
Ota, H. et al. (2007)

J Mol Cell Cardiol 43, 571-9
Sirt1 modulates premature senescence-like phenotype in human endothelial cells
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HUVECin vitroDNAjetPEI-HUVEC
Ota, H. et al. (2009)

J Am Coll Cardiol 53, 2298-305
Sirolimus and everolimus induce endothelial cellular senescence via sirtuin 1 down-regulation: therapeutic implication of cilostazol after drug-eluting stent implantation
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HUVECin vitroDNAjetPEI-HUVEC
Ota, H. et al. (2010)

Arterioscler Thromb Vasc Biol 30, 2205-11
Induction of endothelial nitric oxide synthase, SIRT1, and catalase by statins inhibits endothelial senescence through the Akt pathway
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HUVECin vitrosiRNAINTERFERin
Ou, H. C. et al. (2012)

J Vasc Surg 55, 1104-15
Aspirin prevents resistin-induced endothelial dysfunction by modulating AMPK, ROS, and Akt/eNOS signaling
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C2C12, NIH/3T3in vitroDNAjetPEI
Ounzain, S. et al. (2008)

BMC Mol Biol 9, 50
Comparative in silico analysis identifies bona fide MyoD binding sites within the Myocyte stress 1 gene promoter
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